Why Long COVID is looking more and more like it's driven by 'long infection'
Why Long COVID is looking more and more like it's driven by 'long infection'
Researchers have proposed several biological mechanisms to explain Long COVID. Here's what we know.
Does anyone think we could use a aushealth community for these kinds of posts? It's not news or politics so I didn't really have anywhere else to post it.
It’s not news
Isn't it?
It's filed under analysis. I think of news as stuff that is 'breaking news' even if it's old news. This is kind of that but it's also a brief analytical report. Anyway, maybe I'll just use news next time.
I mean, you could also post this article in the health community of any other instance. It's not exactly specific to Australia...
Maybe an Aus Science community would be good and could include health stuff?
I posted an article to !austech@aussie.zone a while back (https://aussie.zone/post/14355715) which may have fit better in a science comm (but again, could also have gone in plain 'news').
I'm not sure this instance is active enough to stress about which community a post goes, so I think posting here is fine.
Since relatively early in the COVID-19 pandemic, there has been a recognition that in some people, SARS-CoV-2 — or at least remnants of the virus — could stay in various tissues and organs for extended periods. This theory is known as "viral persistence".
While the long-term presence of residual viral fragments in some people's bodies is now well established, what remains less certain is whether the live virus itself, not just old bits of virus, is lingering — and if so, whether this is what causes Long COVID.
This distinction is crucial because live viruses can be targeted by specific antiviral approaches in ways that "dead" viral fragments cannot.